Discovery of biomarkers predictive of GSI response in triple-negative breast cancer and adenoid cystic carcinoma.

نویسندگان

  • Alexander Stoeck
  • Serguei Lejnine
  • Andrew Truong
  • Li Pan
  • Hongfang Wang
  • Chongzhi Zang
  • Jing Yuan
  • Chris Ware
  • John MacLean
  • Philip W Garrett-Engele
  • Michael Kluk
  • Jason Laskey
  • Brian B Haines
  • Christopher Moskaluk
  • Leigh Zawel
  • Stephen Fawell
  • Gary Gilliland
  • Theresa Zhang
  • Brandon E Kremer
  • Birgit Knoechel
  • Bradley E Bernstein
  • Warren S Pear
  • X Shirley Liu
  • Jon C Aster
  • Sriram Sathyanarayanan
چکیده

UNLABELLED Next-generation sequencing was used to identify Notch mutations in a large collection of diverse solid tumors. NOTCH1 and NOTCH2 rearrangements leading to constitutive receptor activation were confined to triple-negative breast cancers (TNBC; 6 of 66 tumors). TNBC cell lines with NOTCH1 rearrangements associated with high levels of activated NOTCH1 (N1-ICD) were sensitive to the gamma-secretase inhibitor (GSI) MRK-003, both alone and in combination with paclitaxel, in vitro and in vivo, whereas cell lines with NOTCH2 rearrangements were resistant to GSI. Immunohistochemical staining of N1-ICD in TNBC xenografts correlated with responsiveness, and expression levels of the direct Notch target gene HES4 correlated with outcome in patients with TNBC. Activating NOTCH1 point mutations were also identified in other solid tumors, including adenoid cystic carcinoma (ACC). Notably, ACC primary tumor xenografts with activating NOTCH1 mutations and high N1-ICD levels were sensitive to GSI, whereas N1-ICD-low tumors without NOTCH1 mutations were resistant. SIGNIFICANCE NOTCH1 mutations, immunohistochemical staining for activated NOTCH1, and HES4 expression are biomarkers that can be used to identify solid tumors that are likely to respond to GSI-based therapies.

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Triple-Negative Breast Cancer and Adenoid Cystic Carcinoma Discovery of Biomarkers Predictive of GSI Response in

Next-generation sequencing was used to identify Notch mutations in a large collection of diverse solid tumors. NOTCH1 and NOTCH2 rearrangements leading to con-stitutive receptor activation were confi ned to triple-negative breast cancers (TNBC; 6 of 66 tumors). TNBC cell lines with NOTCH1 rearrangements associated with high levels of activated NOTCH1 (N1-ICD) were sensitive to the gamma-secreta...

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عنوان ژورنال:
  • Cancer discovery

دوره 4 10  شماره 

صفحات  -

تاریخ انتشار 2014